Recent advances in ankylosing spondylitis: understanding.
Diagnoses of Ankylosing Spondylitis, involves Complete blood count which can help identify if there’s a presence of infection, an Erythrocyte sedimentation rate, which indirectly measures inflammation in the body, X-ray of the spine and pelvis, an HL-B27 antigen test, which looks for any presence of the protein, and last but not the least a physical assessment, which may present tenderness.
Bennett DL. Spondyloarthropathies: Ankylosing Spondylitis and psoriatic arthritis. Radiol Clin North Am 2004. 42(1):121-134. Brower AC. Arthritis: in Black and White. Hardcover. Second edition 1996. Buchmann RF. Imaging of articular disorders in children. Radiol Clin North Am 2004. 2004.
Ankylosing spondylitis (AS) is an inflammatory condition that, if left untreated, can result in the fusing of some of the vertebrae in your spine. When your vertebrae fuse, your spine is less flexible leading to pain, inflammation, reduced posture, difficulty breathing and a reduced quality of life.
Ankylosing spondylitis (AS) is a potentially disabling form of seronegative spondyloarthritis. The main symptom of AS is inflammatory spinal pain; with time, some patients develop ankylosis and spinal immobility. The pathology mainly affects the entheses, where ligaments, tendons and capsules are attached to the bone. Three processes are observed at the entheses: inflammation, bone erosion and.
This article provides an overview of ankylosing spondylitis, including signs and symptoms, diagnosis and management. The article focuses on the difficulties and delays associated with diagnosing this chronic inflammatory disease and developments in diagnostic criteria. Changes in the management of patients with the disease are also discussed, particularly in light of anti-tumour necrosis.
Introduction. Ankylosing Spondylitis (AS) is the major subtype and a main outcome of an inter related group of inflammatory rheumatic disease (spondyloarthropathy) now named spondyloarthritides (SpA) (Lancet, 2007) showing familial aggregation, arthritis of sacroiliac and peripheral joints with enthesopathy, high association with HLA B27 and absence of rheumatoid factor (Brown et al., 1997.
The symptoms of ankylosing spondylitis (AS) usually develop slowly over several months or years. The symptoms may come and go, and improve or get worse, over many years. AS usually first starts to develop between 20 to 30 years of age.